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R This is one mechanism linking slow acetylator status to increased bladder and colorectal cancer risk

Impaired insulin signaling, diminished mitochondrial oxidative capacity, lipid peroxidation, ferroptosis, and microbial dysbiosis not only exacerbate hepatic steatosis but also facilitate the progression from simple steatosis to more severe forms of the disease, such as NASH and cirrhosis [51,56,63,64,65,66,67]

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